1. Muted final journey, Latest Others News - The New Paper
4 mei 2014 · Taiwanese model dies in car crash ... In life, Taiwanese model Chen Ren-Yi posted thousands of photographs on Instagram and Facebook. But in death ...
In life, Taiwanese model Chen Ren-Yi posted thousands of photographs on Instagram and Facebook. But in death, there was no obituary photo of her accompanying her casket at...
2. Ren Cheng Wei (任程伟) - MyDramaList
Bevat niet: injury | Resultaten tonen met:injury
He is a Chinese actor who was born in Heilongjiang province. He graduated from the Shanghai Theatre Academy.
3. Hippocampal excitation-inhibition balance underlies the 5-HT 2C ...
We revealed that inhibition of 5-HT 2C R induced depressive-like behaviours, reduced GABA release and shifted the E/I balance towards excitation in CA3 ...
Shi et al. show that blocking the 5-HT2C receptor induces depression-like behaviours in mice by reducing GABA release, shifting the excitation-inhibition b
4. Tales of a hero to catch the young | Shanghai Daily
27 aug 2009 · Scene from "My Beloved China" portraying the hero of China's revolution, Fang Zhimin, starring Ren Chengwei (left). THOUGH mainstream ...
THOUGH mainstream traditional patriotic films find it hard to attract young audiences these days, film director Hu Xueyang wants to change all that with his latest work,
5. Expression of MANF and NF-κB p65 in cerebral cortex cerebral ischemia ...
... injury in rats[J]. Journal of Bengbu Medical University, 2023, 48(9): 1179 ... PAN Qiong, REN Kai, CHONG Dian-long, ZHANG Zhi-rui, ZHOU Can, LIU Hao ...
ObjectiveTo investigate the role of expression and changes of mesencephalic astrocyte-derived neurotrophic factor (MANF) and nuclear factor (NF)-κB p65 in cerebral cortex of rats with cerebral ischemia-reperfusion injury (CIRI). MethodsThirty adult male SD rats were divided into Sham group and cerebral ischemia-reperfusion group (I/R group) according to random number table, 15 rats in each group.The model of focal CIRI in rats was established by modified thread embolization methods.In the Sham group, the thread embolization was not inserted into the internal carotid artery, and the other treatments were the same as the I/R group.Behavioral changes were observed by neurological deficit score 24 h reperfusion after 2 h ischemia, cerebral infarction area was observed by cranial magnetic resonance (MRI) and TTC staining, and cerebral cortex histopathological changes and cell damage in ischemic area were observed by HE staining.The relative protein levels of MANF and NF-κB p65 were detected by Western blotting, and the protein expression levels of NF-κB p65, CHOP and MANF were detected by immunohistochemistry.The subcellular localization of MANF and NF-κB p65 were observed by immunofluorescence staining.The apoptosis of cerebral cortex cells in the ischemic area was observed by TUNEL method, and the levels of TNF-α, IL-1β and IL-6 in serum of rats were detected by ELISA. ResultsCompared with the Sham group, the rats in the I/R group sh...
6. THE FIRST AFFILIATED HOSPITAL,ZHEJIANG ...
Pain Management; Pathology; Pediatrics; PET Center; Plastic Surgery ... REN Guoping; REN Jingjing; REN Wen; REN Yanling; RUAN Bing; RUAN Jian; RUAN Liming ...
The National Performance Review of Public Hospitals by National Health Commission
7. Chengwei Pan | Papers With Code
13 Oct 2024 • Xinyuan Wang, Victor Shea-Jay Huang, Renmiao Chen, Hao Wang, Chengwei Pan, Lei Sha, Minlie Huang ... injury (TBI). Decision Making · Paper · Add ...
Papers by Chengwei Pan with links to code and results.
8. Protective Effects of Hesperetin Against Lipopolysaccharide ...
28 jun 2022 · Chunzhi G, Zunfeng L, Chengwei Q, Xiangmei B, Jingui Y. Hyperin protects against LPS-induced acute kidney injury by inhibiting TLR4 and NLRP3 ...
Protective Effects of Hesperetin Against Lipopolysaccharide-induced Acute Renal Injury in Rat - Medical Journal of Bakirkoy
9. Expression of antioxidant molecules after peripheral nerve injury ... - OUCI
102395. Scopus. WoS. Crossref citations: 0 · NAD+ metabolism in peripheral neuropathic pain. Yi Dai, Jiaqi Lin, Jinxuan Ren, Bin Zhu, Chengwei Wu, Lina Yu.
AbstractOxidative stress is considered to be one of the main causes of neural damage after injury. However, little is known about the changes in mRNA expression of antioxidant molecules that occur after injury and regeneration of the peripheral nerve. In the present study, the rat median nerve was transected, and transcriptional changes were studied at day 6 and day 12 after injury in both the proximal and the distal stumps, in the absence or presence of microsurgical repair. The expression profiles of the…
10. Vol 45, No 1 (January 2024) - Acta Pharmacologica Sinica
Wei Yu, Lin Wang, Wei-ying Ren, Hai-xia Xu, Ne N. Wu, Dong-hui Yu, Russel J ... PGAM5 exacerbates acute renal injury by initiating mitochondria ...
Acta Pharmacologica Sinica published monthly in English, is the official journal of the Chinese Pharmacological Society and Shanghai Institute of Materia Medica, Chinese Academy of Sciences.
11. Comparative analysis of work-related injury appraisal of 13 cases of ...
PEI Yifei 1, ,; YUAN Wenwen 2, , ,; YANG Lili 3,; JU Huajian 2,; HAN Lu 2. 1. Overseas Business Department, China Railway Fifth Survey and Design Institute ...
Background According to the
Classification and Catalogue of Occupational Diseases , brucellosis is one of the notifiable occupational infectious diseases, which occurs from time to time in the occupational population.Objective To compare the work-related injury appraisal process and results of 13 cases of brucellosis at both provincial and municipal levels, analyze and summarize the bias in the practical work of labor capacity identification for occupational diseases such as brucellosis by appraisal management departments and experts, and propose suggestions for optimizing appraisal work.
Methods A comparative study was conducted on the objective examination results and labor capacity appraisal conclusions based on the occupational contact history, clinical diagnosis, occupational disease diagnosis staging, and labor capacity appraisal of 13 patients with brucellosis. The reasons for the inconsistency between the initial appraisal conclusion by institutions at the municipal level and the final appraisal conclusion by institutions at the provincial level were compared and analyzed.
Results All of the 13 patients with brucellosis applied for municipal-level labor capacity identification after being identified as work-related injuries, 11 of which did not receive a disability level, and 2 were rated as level 10 disability. Four of those who did n...
12. NOX2 inhibitor gp91 ds -tat alleviated lung injury induced by gas ...
... REN Wenjie, YAO Sanqiao. NOX2 inhibitor gp91 ds -tat alleviated lung injury ... YANG Chengwei, ZHANG Jing, LIU Biyong, TANG Yufei, WANG Luhan. Role of ...
Objective To assess the effects of NOX2 (nicotinamide adenine dinucleotide phosphate oxidase) inhibitor gp91 ds-tat on lung injury of rats caused by gas explosion. Methods A total of 90 SD male rats were randomly divided into control group, gas explosion lung injury model group, and NOX2 inhibitor gp91 ds-tat group(n=30/group). The rats in the model group and gp91 ds-tat group were placed in the alleyway 240 m away from the point of detonation. After the explosion, the rats of the gp91 ds-tat group were immediately given once intraperitoneal injection of gp91 ds-tat (1 mg/kg). Then the rats were anaesthetized with blood taken from the abdominal aorta at 24 h, 72 h and 7 d (10 rats in each group) and then executed, respectively. ATP, α -KGDH, ICDHm and PFK were detected in frozen lung tissues. The expression levels of NOX2, CAT protein and mRNA as well as the expression of mitochondrial function -related protein genes were also detected with western blotting and real-time quantitative PCR in rat lung tissues. Results Lung bleeding and collapse were observed in the model group, and the expression of CAT, AMPK, TFAM decreased (P < 0.05), but the expression of NOX2 increased significantly(P < 0.05). Compared with the model group, levels of α-KGDH, ICDHm, PFK and ATP in gp91 ds-tat treatment group increased (P < 0.05), the relative mRNA expression of CAT...
13. DMEM/F12, containing L-glutamide (W/O phenol red)_Cell ... - Solarbio
... Chengwei Wang, Zhou Li, Kailiang Ren. IF：6.3890. Publish_to：Advanced ... injury. Click to check >>. Author：Junming Wan, Hanzhong Liu, Jiachun Li, Yuqing ...
Note：
14. [PDF] Protective Effects of Hesperetin Against Lipopolysaccharide ...
Xu D, Chen M, Ren X, Ren X, Wu Y. Leonurine ameliorates LPS- induced acute kidney injury via suppressing ROS-mediated NF-κB signaling pathway. Fitoterapia ...
15. Journal of Zhejiang University
Author(s):Aidi REN, Chengwei YU, Lixia YANG,... Clicked:956 Download:3120 Cited:0
218. Frontiers of Information Technology & Electronic ... ....
16. Mst1 promotes cardiac ischemia–reperfusion injury by inhibiting the ...
30 jun 2018 · ... Ren K, Gao E, Yang Y, Liang H, Jin Z, Yu S (2017) Melatonin ... Correspondence to Chengwei Zou. Ethics declarations. Conflict of ...
Cardiac ischemia–reperfusion (I/R) injury results mainly from mitochondrial dysfunction and cardiomyocyte death. Mitophagy sustains mitochondrial function and exerts a pro-survival effect on the reperfused heart tissue. Mammalian STE20-like kinase 1 (Mst1) regulates chronic cardiac metabolic damage and autophagic activity, but its role in acute cardiac I/R injury, especially its effect on mitophagy, is unknown. The aim of this study is to explore whether Mst1 is involved in reperfusion-mediated cardiomyocyte death via modulation of FUN14 domain containing 1 (FUNDC1)-related mitophagy. Our data indicated that Mst1 was markedly increased in reperfused hearts. However, genetic ablation of Mst1 in Mst1-knockout (Mst1-KO) mice significantly reduced the expansion of the cardiac infarction area, maintained myocardial function and abolished I/R-mediated cardiomyocyte death. At the molecular level, upregulation of Mst1 promoted ROS production, reduced mitochondrial membrane potential, facilitated the leakage of mitochondrial pro-apoptotic factors into the nucleus, and activated the caspase-9-related apoptotic pathway in reperfused cardiomyocytes. Mechanistically, Mst1 activation repressed FUNDC1 expression and consequently inhibited mitophagy. However, deletion of Mst1 was able to reverse FUNDC1 expression and thus re-activate protective mitophagy, effectively sustaining mitochondrial homeostasis and blocking mitochondrial apoptosis in reperfused cardiomyocytes. Finally, we demonstrat...
